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In the case of glucocorticoids, their effect is produced by the direct action of their receptor on the response elements located at the promoter and first exon level (Figure 7), facilitating the access of other transcription factors (especially Pit-1 and CREB) to their specific binding sequences [42]

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Tables 1 and 2 provide lists of cytokines and growth factors that increase and decrease the intestinal TJ barrier and their molecular mechanisms, respectively