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The possible mechanism is that although CagA induces DNA damage in the GHCs, it does not lead to apoptosis, which greatly aggravates the risk of gastric cancer (Chaturvedi et al., 2011)
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reported that HRS combined with early aerobic exercise enhances acute myocardial infarction-induced superoxide dismutase levels and total antioxidant capacity, promotes mitochondrial and DNA repair by partially regulating the expression of antioxidant-associated proteins and mitochondria-associated proteins, and protects against myocardial injury after MI [85]
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