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3.11 Neuroprotective efficacy of GO@GSH-FA on neuronal injury post-ischemia-reperfusion To elucidate the neuroprotective potential of GO@GSH-FA, we performed both histological and molecular analyses on cerebral ischemia-reperfusion injury lesions
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Long-term incubation of human erythrocytes with aluminum induced PS externalization, increased intracellular Ca 2+ levels, and degradation of AE1 proteins, which are markers of eryptosis
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